Download Atlas of Esophageal Disease and Intervention: A by Shanda H. Blackmon, Min P. Kim, Karen J. Dickinson PDF

By Shanda H. Blackmon, Min P. Kim, Karen J. Dickinson

ISBN-10: 1493930877

ISBN-13: 9781493930876

ISBN-10: 1493930885

ISBN-13: 9781493930883

This atlas offers a entire, cutting-edge assessment of all interventions that pertain to the esophagus. It features a evaluation of the present staging modalities, ablation applied sciences, resection and reconstruction thoughts, and ailment type. Evidence-based directions relating to how every one intervention is selected also are incorporated. With colour illustrations and images for every surgical procedure, the atlas information particular anatomic issues reminiscent of micro-anatomy of Barrett’s and Dysplasia, EMR pathology, endoscopic ultrasound, and standard surgical anatomy. every one intervention is gifted in job layout as a job record to be checked-off as every one step is completed.

Written through specialists within the box, Atlas of Esophageal illness and Intervention: A Multidisciplinary Approach serves as a invaluable source for any practitioner who plays esophageal intervention and should consultant new surgeons and gastroenterologists into the hybrid multidisciplinary method of this disease.

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Extra resources for Atlas of Esophageal Disease and Intervention: A Multidisciplinary Approach

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14 (a, b) Esophageal squamous cell carcinoma, gross photos 2 Histologic Anatomy Fig. 15 (a) Squamous cell carcinoma (H & E, ×40). This moderately differentiated squamous cell carcinoma involves the full thickness of the biopsy specimen. Tumor necrosis is prominent. (b) Squamous cell carcinoma (H & E, ×200). The tumor cells have enlarged hyperchromatic nuclei and infiltrate the stroma in irregular nests. (c) Squamous cell carcinoma (H & E, ×400). The presence of intercellular bridges (delicate filaments seen between the tumor cells) indicates squamous differentiation, as does keratin production 39 a b c 40 B.

By definition, a diagnosis of mucinous carcinoma is rendered when more than 50 % of the lesion consists of mucin [7]. 34 Fig. 10 (a) Intramucosal adenocarcinoma (H & E, ×100). (b) Intramucosal adenocarcinoma (H & E, ×200) B. Gorman a b Fig. 11 Untreated adenocarcinoma at the gastroesophageal junction (fresh specimen) 2 Histologic Anatomy a 35 b c Fig. 12 Esophageal adenocarcinoma. (a) Adenocarcinoma (H & E, ×20). (b) Adenocarcinoma showing perineural invasion (H & E, ×100). (c) Adenocarcinoma with complex, cribriform architecture (H & E, ×400).

The mucosa on the right shows normal esophageal squamous epithelium. (b) Inlet patch (H & E, ×200). On higher power, the gastric mucosa of the inlet patch is identical to that seen in the stomach, with foveolar epithelium overlying gastric glands composed of mucus neck cells, parietal cells, and chief cells. Oxyntic-type gastric mucosa is most common, although antral and mixed type are also seen [10] 46 B. Gorman Achalasia Achalasia, the failure of smooth muscle fibers (usually in the esophagus and lower esophageal sphincter) to relax, can cause dilatation of the distal esophagus.

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Atlas of Esophageal Disease and Intervention: A Multidisciplinary Approach by Shanda H. Blackmon, Min P. Kim, Karen J. Dickinson


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